IndraLab

Statements

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"HCQ reduces proinflammatory cytokines such as IL-1, IL-6 and tumor necrosis factor-alpha, and has also been shown to interfere with antigen processing and presentation and to work synergistically with the calcineurin inhibitors to suppress proliferative T-cell responses in vitro [XREF_BIBR, XREF_BIBR]."
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"Thus, HCQ therapy inhibits the production of tumor necrosis factor-alpha (TNF-alpha), IL-1, and IL-6 by mononuclear cells [24,27]."
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"Moreover, chloroquine (CQ), the precursor for HCQ, is found to decrease the production of Tumor necrosis factor-alpha (TNF-α), IL-1 beta (β) and IL-6 from lipopolysaccharide (LPS)-stimulated human monocytes (Jang et al., 2006)."
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"In addition, HCQ can reduce the production of tumour necrosis factor-α, interleukin (IL)-17, IL-6, IL-1β and IL-18, and other cytokines by affecting macrophages and monocytes [36]."
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"HCQ reversed the sepsis-induced increase in malondialdehyde, tumor necrosis factor-alpha, interleukin-6, and lactic acid."
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"Hydroxychloroquine also inhibited CpG induced production of interleukin-6 and tumor necrosis factor-alpha in B cell subsets."
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