IndraLab

Statements

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"Similar strategies could be employed to uncover USP47 substrates in hepatocytes in the context of liver regeneration to provide more mechanistic insights for this process.Another caveat in our proposa[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"At this point, the mechanism whereby USP47 contributes to liver regeneration is not clear."
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"Because inflammasome-mediated processing of IL-1β and IL-18 has been shown to be indispensable for liver regeneration in a model of acute liver failure [ 57 ], it is possible that USP47 may contribute[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Therefore, we hypothesized that USP47 might contribute to liver regeneration by regulating proliferation of hepatocytes."
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"USP47 deletion or inhibition dampens liver regeneration whereas USP47 over-expression enhances liver regeneration in mice.All the animal experiments were reviewed and approved by the Nanjing Medical U[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Despite these observations, our finding adds to the growing list of genes targeted by E2F1 during liver regeneration.Second, we show that USP47 deletion or inhibition attenuates whereas USP47 over-exp[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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