IndraLab

Statements

USP14 inhibits Cell Survival. 9 / 9
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"To determine the mode of cell death involved in the reduction of osteosarcoma cell viability by TGT, we treated 143B and SAOS2 cells with TGT at concentrations of 0, 25, 50, and 75 mg/mL, followed by staining using APC Annexin V in combination with 7-AAD."
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"Knock-down of USP14 in multiple myeloma cells induces loss of cell viability."
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"Moreover, TGT and mL-098 (an activator of RAS) co-treatment reduced AML12 cell viability via regulating autophagy and TGT-induced liver injury-related indicators more dramatically than TGT treatment alone, whereas Salirasib (an inhibitor of RAS) had an opposite effect."
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"In renal epithelial cells subjected to hypoxia/reoxygenation (H/R), Usp14 knockdown increased cell viability and reduced apoptosis."
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"Pharmacologic inhibition of USP14 using b-AP15 leads to apoptosis in several human cancer cell lines by decreasing cell viability and increasing ROS generation [[298], [299], [300]].9 Future directions and conclusion."
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"Interference of USP14 suppressed MCL cell viability, potentiated cell cycle arrest, apoptosis, and ibrutinib sensitivity."
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"USP14 and UCHL5 short interfering RNA knockdown decreases MM cell viability."
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"Similarly , UbVs were generated with high affinity for USP14 , which inhibited Ub signaling and cell survival in yeast [ 106 ] ."
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"Similarly, UbVs were generated with high affinity for USP14, which inhibited Ub signaling and cell survival in yeast [XREF_BIBR]."
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