IndraLab

Statements

KCNH2 activates cell adhesion. 7 / 8
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"The blockage of hERG1 Khori channels impaired the ability of MVs to induce leukemia-cell chemotaxis and strikingly inhibited the adhesion of leukemia cells to HUVECs."
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"Another mechanism involves bile acid-activated proinflammatory mediators including early growth response factor-1 (Erg-1), which stimulates the pro-inflammatory signaling and effector including cytoki[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"HERG K+ channels activation during beta (1) integrin mediated adhesion to fibronectin induces an up-regulation of alpha (v) beta (3) integrin in the preosteoclastic leukemia cell line FLG 29.1."
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"We have recently shown that hERG1 is physically linked to beta1 integrins and thereby modulate adhesion dependent signaling (Cherubini et al, 2005)."
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"HOFMANN G, BERNABEI PA, CROCIANI O et al. : HERG K+ channels activation during beta1 integrin mediated adhesion to fibronectin induces an up-regulation of alphavbeta3 integrin in the preosteoclastic leukemia cell line FLG 29.1."
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"Finally, the beta1 integrin subunit, K V 11.1 and NHE1 co-immunoprecipitated in colorectal cancer cells seeded onto Collagen I, suggesting the formation of a macromolecular complex following integrin-mediated adhesion."
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"Moreover, K v 3.3 may also be involved in the cell adhesion process, similar to the function of hERG potassium channels [XREF_BIBR], even if the effect is in the opposite direction; the hERG potassium channels enhance cell adhesion, whereas K v 3.3 inhibits cell adhesion."
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