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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP17L2 deubiquitinates H2AX. 5 / 5

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ubibrowser

"Dub3 controls DNA damage signalling by direct deubiquitination of H2AX"

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"Overexpression of DUB3 decreased monoubiquitination of γH2AX in the absence of an exogenous DNA damage source as well as post-IR [33]."

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reach

"Moreover, Dub3 and H2AX interact and Dub3 deubiquitinates H2AX in vitro."

24704006

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"Moreover, Dub3 and H2AX interact and Dub3 deubiquitinates H2AX in vitro."

24704006

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"DUB3 (also known as USP17L2) directly interacts with and deubiquitylates H2AX [73]."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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USP17L2 deubiquitinates H2AX. 5 / 5

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