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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

NFATC3 activates KCNMA1. 2 / 2

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"AKAP150 also participates in NFATc3-mediated BK channel downregulation in HFD-induced diabetic mice (Figure 5; Nystoriak et al., 2014)."

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"These results support a role for the transcription factor NFATc3 in regulating UBSM contractility, primarily through an NFATc3 dependent increase in BK channel activity."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...