IndraLab

Statements

USP9X activates BIRC5. 3 / 3
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"USP9X WT markedly impeded survivin degradation by IITZ-01 in Caki-1/Vector cells, whereas USP9X/C1566S still revealed IITZ-01-inhibited survivin expression (Figure 6e)."
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"Conversely, USP9X is known to promote the activities of anti-apoptotic factors, MCL1 and Survivin."
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"We transfected USP9X shRNAs into PLC/PRF/5 cells with LNC473 overexpression, and found that depletion of USP9X abolished the upregulation of survivin mediated by LNC473 overexpression ( Fig. 5 B)."
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