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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP40 activates CFLAR. 5 / 5

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"Consequently, USP40 augmented the stability of CFLAR via its de-ubiquitinase activity."

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"These findings demonstrate USP40 promotes the stability of CFLAR at the protein level."

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"Using a reverse experiment in A549 cell lines, we showed that USP40 overexpression slightly increased CFLAR protein when GMEB1 was knocked down (Fig. 5e)."

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"USP40 targeted CFLAR L for K48 linked de-ubiquitination."

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"We found that GMEB1 promoted the stability of CFLAR by de-ubiquitinase USP40."

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USP40 activates CFLAR. 5 / 5

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