IndraLab

Statements

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"Then in 2011, they found that E2 reduced the currents with Ussing chamber mediated by the KCNQ1 : KCNE3 potassium channel rather than KCNQ1 : KCNE1 or KCNQ1 alone [XREF_BIBR]."
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"The E2 inhibition of toxin induced intestinal secretion occurs via sex-specific PKCδ dependent modulation of KCNQ1:KCNE3 channels to reduce the driving force for Cl − secretion [61] ."
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"Patch-clamp experiments in CHO cells have demonstrated the molecular mechanism for E2 inhibition of KCNQ1:KCNE3 channel current via PKCδ phosphorylation of KCNE3 at the S82 amino acid site [59] with t[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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