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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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Statements

COPS6 activates epithelial to mesenchymal transition. 5 / 5

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"In HCC, CSN6 overexpression promotes epithelial-mesenchymal transition and predicts poor prognosis (26)."

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"Additionally, CSN6 was found to promote EMT by inhibiting E-cadherin, which were significantly mitigated via upregulation of Snail as a result of MEK and ERK pathway activation."

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"CSN6 promotes PTC progression by inducing the EMT."

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"CSN6 induces EMT and enhances metastasis of breast cancer cells by reducing Snail1 ubiquitination ."

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"Together, the results suggest that CSN6 may positively regulate beta-catenin transcription, thereby mediating the EMT."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

COPS6 activates epithelial to mesenchymal transition. 5 / 5

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