IndraLab

Statements

EIF3H activates HYCC1. 10 / 10
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"EIF3H depletion inhibits O-GlcNac pathway activity in HCC."
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"EIF3h is upregulation in the breast cancer, HCC, lung cancer, and colon cancer et al [ 2 , 99 , 100 ]."
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"EIF3H enhances the stability of OGT in HCC."
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"Finally, EIF3H could promote cancer progression in HCC via OGT."
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"EIF3H promotes HCC progression in vitro and in vivo."
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"Our results showed that the depletion of EIF3H markedly decreased the proliferation and migration of HCC both in vitro and in vivo."
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"In addition, we also confirmed that EIF3H promoted HCC cell proliferation and migration via upregulation of OGT."
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"The results illustrated that EIF3H depletion by lentivirus-based shRNA suppressed the HCC tumor growth markedly (Fig. 4G, H)."
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"Moreover, we also demonstrated that EIF3H may accelerate the HCC progression via the regulation of OGT expression and ferroptosis."
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"EIF3H has been found to promote HCC progression by stabilizing OGT (O-GlcNAc transferase) and inhibiting ferroptosis, a form of regulated cell death."
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