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"In heterologous expression systems, DPP10 interacts with Kv4.3 channels, shifting the voltage-dependence of steady-state activation and inactivation to more negative potentials as well as accelerating[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

sparser
"Within the N-glycosylation mutations, N257Q showed the most pronounced effects with an impaired membrane trafficking completely disrupting the interaction between Kv4.3 and DPP10."