IndraLab

Statements

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"The overexpression of OTUB2 promoted glycolysis and induced the proliferation, migration, and invasion of endometrial cancer cells."
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"These results indicated that OTUB2-regulated expression of CTNNB1 could enhance ZEB1 to promote the invasion and metastasis of iCCA cells.To further investigate the mechanism of CTNNB1 upregulation in[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Moreover, knockdown of OTUB2 significantly suppressed the invasion and migration abilities of A549 and H1299 cells."
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"We also confirmed that OTUB2 enhanced the proliferation, migration, and invasion abilities of NSCLC cells In vitro."
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"In contrast, stable overexpression of OTUB2 significantly increased the invasion and migratory abilities of XL-2 and H292 cells."
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"Overexpression of OTUB2 enhanced glycolysis and promoted cell growth, migration, and invasion via the AKT/mTOR signaling pathway in A549 and H1299 cells and a xenograft mouse model (Li J. et al., 2019)."
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"These results indicate that OTUB2-regulated expression of ZEB1 enhances EMT to promote invasion and metastasis in iCCA cells ( Fig. 4 E and F)."
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"Meanwhile, in vitro experiments confirmed that the reduction in cell migration and invasion ability caused by low OTUB2 expression could be attenuated by CTNNB1 overexpression."
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