IndraLab

Statements

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"Knockdown of USP12 inhibited HCC cell proliferation and promoted apoptosis via the p38-MAPK pathway."
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"Additionally, only the overexpression of USP12 promoted cell cycle progression (Fig. 3F, G) and inhibited apoptosis (Fig. 3H, I), whereas USP12 did not."
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"Depletion of USP12 decreased PC cell proliferation, increased apoptosis and caused G 1 arrest ( Burska et al., 2013 and McClurg et al., 2014 )."
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"In addition, USP12 knockdown increased apoptosis in HCC cell lines."
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"We further established that Usp12 depletion reduced PC cell proliferation and induced apoptosis, furthermore Usp12 levels are increased in PC tissue [XREF_BIBR]."
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"Besides, flow cytometry apoptosis Annexin V-PI staining assay indicated that USP12 deletion promoted apoptosis, and YAP overexpression was able to rescue this effect (Fig. 5I, J)."
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