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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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ATXN3L decreases the amount of KLF5. 3 / 3

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"Knockdown of ATXN3L decreased KLF5 protein levels and inhibited the proliferation of breast cancer cells [XREF_BIBR]."

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"Nevertheless, ATXN3L increased the KLF5 protein stability and inhibited the expression of KLF5 downstream target genes, such as p21 and p27, and promote basal type breast cancer cell proliferation."

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"ATXN3L was identified as a candidate KLF5 DUB because the siRNA pool, and 2 out of 3 individual siRNA, against ATXN3L dramatically decreased the KLF5 protein level in HeLa."

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ATXN3L decreases the amount of KLF5. 3 / 3

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