IndraLab

Statements

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"In summary, these data indicate that USP4 enhances HCC cell proliferation, migration, and invasion and suppresses HCC cell apoptosis in vitro."
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"XREF_BIBR also demonstrated that inhibiting USP4 expression in GBM cells both inhibited their proliferation and induced apoptosis via antagonizing USP4 mediated stimulation of the ERK signaling pathway."
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"In this study, we demonstrated that USP4 negatively regulated RIP1-mediated NF-κB activation and promoted TNF-α-induced apoptosis through interacting with RIP1 and deubiquitinating K63-linked ubiquiti[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Recent findings indicate that USP4, upregulated in TMZ-resistant GBM cells, inhibits apoptosis in a p53-dependent manner, and this resistance is further amplified by p53-specific inhibitors [103]."
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"In addition, Overexpression of USP4 reduced hepatic inflammatory infiltration and apoptosis upon HIR stimulation."
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"We then tested whether USP4 expression inhibits cell apoptosis upon DNA damage."
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"It was found that over-expression of USP4 enhanced the ability of RD cells to fight against invading EV71, and also reduced cell apoptosis."
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"Additionally, USP4 overexpression inhibited hepatocyte inflammation and apoptosis on hepatic I/R stimulation."
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"The results demonstrated that stable knockdown of USP4 significantly enhanced the ability of poly (I:C) to induce apoptosis ( Fig. 4 B–C), while stable overexpression of USP4 markedly attenuated the p[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"METTL3 elevates the levels of oncogenes such as THRAP3, RBM25, USP4, and BZW2, which accelerate the proliferation of MM cells and inhibit apoptosis [233, 234]."
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"Stable knockdown of USP4 significantly enhanced the ability of diABZI to induce apoptosis ( Fig. 4 F–G), whereas stable overexpression of USP4 but not USP4 C311A markedly attenuated diABZI-induced apo[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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