IndraLab

Statements

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"We also demonstrated that VCPIP1 overexpression promoted the HBV cccDNA transcription and enhanced the HBV gene expression in the HBV-infected HepG2-NTCP cells."
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"By using a series of HBV replication and infection models, we extensively characterized VCPIP1-mediated upregulation of HBV transcription, antigen expression, and genome replication."
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"VCPIP1 enhances HBV transcription, antigen expression, and genome replication."
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"In the recent report by Wu et al., overexpression of VCPIP1 and/or PSMC3 was also shown to cause increased HBV transcription, antigen secretion, and progeny virus production in infected HepG2-NTCP cells."
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"The recent report by Wu et al. also showed that overexpression of VCPIP1 and/or PSMC3 increased HBV transcription, antigen secretion, and progeny virus production in infected HepG2-NTCP cells (17)."
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