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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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KCNMA1 activates acetylcholine. 3 / 3

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"Local administration of a BK channel blocker iberiotoxin (2 microM) through a dialysis fiber increased the stimulation induced ACh release from 7.6 +/-2.7 to 9.0 +/-3.2 nM (P < 0.05)."

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"Not surprisingly, application of the BK channel blockers IBTX (100 nM) or CHTX (1 microM) only slightly reduced the amplitude and duration of IK, ACh in apical OHCs (XREF_FIG)."

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"Furthermore, vessel experiments demonstrated that another BK channel blocker CTX could enhance ACh stimulated vasoconstrictions in the umbilical veins."

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Statements

KCNMA1 activates acetylcholine. 3 / 3

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