IndraLab

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"However, in the context of cisplatin intervention, OTULIN knockdown markedly enhanced the inhibitory effect of cisplatin on proliferation (Fig. 2a, b)."
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"OTULIN is critical for restraining life threatening spontaneous inflammation, maintaining immune homeostasis and activating NF-kappaB to promote the secretion of pro inflammatory cytokines and restricts bacterial proliferation in infection [XREF_BIBR, XREF_BIBR]."
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"This scenario was later confirmed by reports showing that OTULIN deletion in the liver causes TNFR1-driven, apoptosis- and compensatory proliferation-mediated liver pathology, while OTULIN deletion in keratinocytes causes TNFR1-driven, RIPK1 kinase activity-mediated, cell death-dependent skin inflammation [65,66] (Table 1)."
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