IndraLab

Statements



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"OTUB1 promotes CRC metastasis by facilitating EMT and acts as a potential distant metastasis marker and prognostic factor in CRC."

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"The results confirmed that OTUB1 promoted EMT in endometriosis , which is consistent with our previous findings ( Figures 8G and 8H ) ."

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"In several cancers, OTUB1 promoted cancer development through facilitating EMT 14, 20-22."

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"In addition, OTUB1 enhances glycolysis and epithelial-mesenchymal transition of endometriosis cells, leading to promote proliferation, migration, and invasion of endometriosis cells."

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"Together, OTUB1 promotes epithelial-mesenchymal transition in endometriosis cells."

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"They revealed that OTUB1 knockdown significantly inhibited tumor growth.Furthermore, western blot analysis revealed that OTUB1 promoted GBM through facilitating EMT."

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"Similar findings were reported by Xu et al., showing that OTUB1 knockdown inhibited EMT in GBM 22.In exploring the regulatory mechanism of OTUB1, we found that it promotes GBM growth by inhibiting the JAK2/STAT1 signaling pathway."

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"The results confirmed that OTUB1 promoted EMT in endometriosis, which is consistent with our previous findings (Figures 8G and 8H)."

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"OTUB1 promotes colorectal cancer metastasis by facilitating EMT and acts as a potential distant metastasis marker and prognostic factor in CRC [39]."

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"Our data showed that overexpression of OTUB1 altered the expression levels of EMT-related factors and promotes EMT in endometriosis cells."

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"OTUB1 promote the migration and invasion of CRC cells in vitro by altering EMT markers."

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"OTUB1 also promoted the metastasis of CRC cell lines in vitro and in vivo by regulating EMT."