IndraLab

Statements

reach
"All-trans-retinoic acid inhibits Jun N-terminal kinase dependent signaling pathways."
9507016
reach
"ATRA can inhibit the activator protein-1 (AP-1) activity in ATRA sensitive cell lines, but not in ATRA resistant cell line, and the anti-AP-1 activity of ATRA is mediated by its receptor, retinoic acid receptor alpha (RAR alpha)."
11776129
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"All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity."
10022884
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"All-trans-retinoic acid (t-RA) inhibited Jun N-terminal kinase (JNK) and, to a lesser extent, extracellular signal regulated kinase activity in normal HBE cells."
9507016
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"In contrast, ATRA decreased the phosphorylated form of c-Jun in the absence of HCV Core."
29156743
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"The changes in the levels of the following proteins were confirmed by conventional western immunoblotting : all-trans-retinoic acid increased ELF3, topoisomerase II alpha, RB2 and p130, RIG-G, and EMAPII and decreased MEF2D and cathepsin L. N-(4-Hydroxyphenyl) retinamide up-regulated ELF3, c-Jun, Rb2 and p130, JAK1, p67phox, Grb2, O (6)-methylguanine-DNA methyltransferase, and Ercc-1."
15059897
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"ATRA is a potent inhibitor of activator protein 1, a transcription factor of the JNK pathway."
18720064