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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP14 activates endoplasmic reticulum. 3 / 3

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"At the same time, liver specific knockout of USP14 eliminated the effect of ER stress on glucose metabolism and also improved hyperglycemia and glucose intolerance in obese mice."

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"Collectively, we suggest that inhibition of USP14 and UCHL5 potently blocks ERα signaling via downregulating the expression and transcriptional activity of ERα."

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"USP14 and UCHL5 inhibitors suppress the growth of ER + BCa cells."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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USP14 activates endoplasmic reticulum. 3 / 3

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