IndraLab

Statements

USP2 activates USP2. 3 / 4
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"On the other hand, a defect of peroxisomal protein import or even release of USP2 from defective peroxisomes because of cell injury or dysfunction would increase the cytosolic pool of USP2, which might contribute to the initiation of apoptosis."
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"Altogether, we initially demonstrated that USP2 overexpression suppressed hypertrophy, calcium overload, and mitochondrial dysfunction and increased protein levels of MFN2 via deubiquitination in Ang [MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"To further investigate the potential involvement of USP2 in cisplatin resistance in NSCLC through ferroptosis, USP2 expression was overexpressed and the impact on cellular ferroptosis was assessed."
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