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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP18 inhibits NOTCH1. 3 / 3

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"Second, USP18 overexpression reduces the K48 linked ubiquitination level of Notch1, whereas USP18 silencing significantly increases the Notch1 K48 linked ubiquitination."

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"Moreover, USP18 overexpression reduced the K48 linked ubiquitination level of Notch1, whereas USP18 silencing significantly increased Notch1 K48 linked ubiquitination (XREF_FIG - XREF_FIG)."

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"Indeed, we found here that USP18 silencing could increase the turnover rate of Notch1 in the presence of cycloheximide (CHX) (XREF_FIG and XREF_FIG)."

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Statements

USP18 inhibits NOTCH1. 3 / 3

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