IndraLab

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USP13 activates ATG5. 7 / 7
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"On the contrary, IGF2BP2 elevates the stability of a deubiquitinase ubiquitin specific peptidase 13 (USP13) to prolong the half-life of autophagy-related protein 5 (ATG5), inducing pro-survival autophagy and drug resistance in gastrointestinal stromal tumors 60."
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"Taken together, these findings demonstrated that USP13 upregulated ATG5 via an m A-dependent mechanism through binding with and stabilizing the m A writer METTL3 in OS."
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"USP13 promotes oncogenic autophagy dependent on METTL3-mediated upregulation of ATG5 in OS cells."
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"Collectively, our study results indicate that USP13 promotes glycolysis and tumor progression in OS by stabilizing METTL3, thereby stabilizing ATG5 mRNA and facilitating autophagy in OS."
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"USP13 promotes AML cell growth and autophagy by upregulating ATG5."
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"USP13 promotes acute myeloid leukemia cell proliferation and autophagy by promoting ATG5."
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"In gastrointestinal mesenchymal tumors, inhibitors of USP13 attenuate ATG5 and inhibit m6A modification-mediated autophagy."
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