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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

BK channel inhibitor activates KCNMA1. 2 / 2

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"The rapid effects of 17beta-estradiol on whole cell K + currents were blocked by the BK channel inhibitor paxilline, suggesting a BK channel dependent mechanism."

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"As illustrated in Figure XREF_FIG A, the selective BK channel inhibitor paxilline (1mumol/L) completely abolished single BK channel activity (n = 5, N = 5; P < 0.05), suggesting that the stimulatory effect of 17beta-estradiol on channel NPo was due to BK channel activation."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...