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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP18 activates Pancreatic Neoplasms. 3 / 3

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"Moreover , we found that USP18 promoted pancreatic cancer progression via upregulation of Notch-1-dependent c-Myc ."

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"Overall , these results demonstrated that USP18 contributes to the progression of pancreatic cancer and is dependent on the Notch1 / c-Myc signalling pathway ( Figure 7G ) ."

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"USP18 contributes to the progression of pancreatic cancer through enhancing the Notch1-c-Myc axis USP18 has been reported to interact with different substrates to exert its effects [ 16-18 ] ."

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USP18 activates Pancreatic Neoplasms. 3 / 3

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