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BID binds CACNA1A and BAK1. 1 / 1

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"We further established for the first time that the C-terminal domain of Mcl-1 became proapoptotic as a result of caspase-3 cleavage, and its physical interaction and cooperation with tBid, Bak, and voltage-dependent anion-selective channel 1 promoted mitochondrial apoptosis."

15637055

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IndraLab

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BID binds CACNA1A and BAK1. 1 / 1

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