IndraLab

Statements

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"Our investigation also found that USP3 overexpression suppressed GC cell migration and proliferation, suggesting that USP3 was a miR-224-5p downstream target.In summary, we identified that hsa_circ_00[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Through in vitro cell proliferation (Fig. 2C), colony formation (Fig. 2D), migration (Fig. 2E), wound healing (Supplemental Fig. 1C) and invasion (Fig. 2F) assay, we found that downregulation of USP3 inhibited the proliferation, migration and invasion of osteosarcoma cells."
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"Depletion of tyrosine tRNA GUA or its translationally regulated targets USP3 and SCD repressed proliferation revealing a dedicated tRNA regulated growth-suppressive pathway for oxidative stress response."
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"Knockdown of USP3 in U2OS cells with two different shRNAs significantly enhanced cell proliferation ( Fig. 4 a)."
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"When we reconstitute WT USP3, but not the C168S catalytic-dead mutant USP3 in the knowdown cells, we found that reconstitution of WT USP3 reversed the increased proliferation caused by USP3 deficiency[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Clone formation ( Fig. 5 E–G) and Transwell ( Fig. 5 H–J) assays validated that USP3 upregulation reversed miR-224-5p-induced inhibition of migration and proliferation in MKN-28 and AMGC803 cells.Poor[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"The absence of USP3 in vitro and in vivo models has been shown to decrease NB cell proliferation, migration, and invasion and inhibit tumor growth (43)."
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"Additionally, overexpressed USP3 significantly promoted cell proliferation in vitro and tumor growth in vivo, while the silencing of USP3 inhibited proliferation and tumor growth."
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