Other Services

This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

UCHL1 activates CD36. 3 / 3

| 1 2

➶

reach

"To further explore the molecular mechanism of UCHL1 on the regulation of CD36, we observed the inhibition of UCHL1 promotes CD36 degradation."

32801299

➶

eidos

"Subsequently , we found that UCHL1 inhibitor or siRNA downregulates the expression of CD36 protein whereas SR-A , ABCA1 , ABCG1 , Lox-1 , and SR-B1 have no significant change ."

32801299

➶

reach

"This results indicates that the suppression of lipid accumulation induced by UCHL1 inhibition or silence is a result of UCHL1 inhibition promoted CD36 degradation."

32801299

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

Other Services

This Service

This Project

IndraLab

Statements

UCHL1 activates CD36. 3 / 3

Our Sources:

Databases

Reading