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UCHL1 activates homeostatic process. 2 / 2

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"Compared with the control group, UCHL1 knockout significantly reduced the half-life of the TAZ protein, suggesting that UCHL1 regulates TAZ homeostasis by blocking TAZ proteasome-mediated degradation (Figure 3I)."

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"Collectively, these results indicate that UCHL1 promotes HGSOC growth by mediating protein homeostasis through the PSMA7-APEH-proteasome axis."

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UCHL1 activates homeostatic process. 2 / 2

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