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Calcium(2+) inhibits CASP1. 5 / 6
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"Treatment with 1, 2-bis (2-aminophenoxy) ethane-N, N, N ', N '-tetraacetic acid acetoxymethyl ester, a chelator of intracellular calcium, inhibited IL-32-induced IL-1 beta production and caspase-1 activation."
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"The presence of a Ca chelator, Bapta, prevented the increased dmDNA-induced ROS generation (RFU, dmDNA+Bapta = 980 ± 169 vs. dmDNA = 2380 ± 401; P < 0.05) (Figure 6B) and caspase-1 activation (AU, LPS+dmDNA+Bapta = 0.9 ± 0.2 vs. LPS+dmDNA = 2.6 ± 0.4; P < 0.05) in endothelial cells (Figure 6C)."
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"In addition, our experiments showed that the Ca 2+ chelator, BAPTA-AM, did not completely inhibit caspase-1 activation and mitochondrial ROS production."
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"The lotr1 mutants display altered localization of CASP1, an essential protein for Casparian strip formation [9], disrupted Casparian strips, ectopic suberization of endodermal cells, and low accumulation of shoot calcium (Ca)."
28238658
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"Both ACA, a channel blocker that acts on several transient receptor potential (TRP) channels including TRPM2, and 3MFA, a TRPM2 specifical inhibitor [53,54], significantly suppressed Ca 2+ influx in U[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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