IndraLab

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KCNK2 activates Cell Survival. 3 / 3
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"Our findings suggested that Foxd3 potentiates miR-214-dependent inhibition of Kcnk2 and may in turn contribute to the exacerbation of SCII.Initially, our results showed that Kcnk2 was poorly expressed in SCII and that the overexpression of Kcnk2 enhanced the cell viability and restrained the cell apoptosis of PC12 cells."
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"However, the overexpression of Kcnk2 or knockdown of Foxd3 enhanced the cell viability and inhibited the apoptosis of the hypoxia/reoxygenation-treated PC12 cells (56)."
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"In addition, upregulation of Kcnk2 or knockdown of Foxd3 promoted the cell viability and reduced the apoptosis of the H/R-treated PC12 cells."
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