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"Additionally, PARP1 mediates the effect of USP36 on Dox-induced cardiotoxicity modulation."

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"These data showed that USP36 promotes Dox-induced cardiomyocyte oxidative stress injury and apoptosis via PARP1.To better understand the role and functional significance of USP36 in Dox-induced cardio[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"