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BAP1 inhibits pathogenesis. 1 / 1

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eidos

"More importantly , when compared to NF2 and CDKN2A double knockout tumors where BAP1 expression was retained , many of the top differentially expressed genes in triple knockout tumors were downstream targets of PRC2 , uncovering a connection between BAP1 and PRC2 and thereby confirming in vivo how , at least in part , the loss of BAP1 contributes to MPM pathogenesis [ 204 ] ."

| PMC

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IndraLab

Statements

BAP1 inhibits pathogenesis. 1 / 1

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