IndraLab

Statements

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"CACNA1D mutation (V1153G) causes adrenal cortical proliferation and aldosterone (Ald)-producing adenoma [3]."
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"Similarly, our previous study indicated that calcium channel Cav1.3 increased the proliferation and metastasis in EC cells [ 50 ]."
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"Previous studies have identified somatic mutations in the potassium channel gene KCNJ5 and the calcium channel gene CACNA1D, which contribute to aldosterone production and cell proliferation in adrenal glomerulosa [6, 7]."
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"Moreover , Cav1.3 knockdown markedly impaired estrogen-stimulated calcium influx , cell proliferation , and migration of endometrial cancer cells ."
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"CACNA1D may simultaneously promote PCa cell growth and proliferation [XREF_BIBR], and thus play a generalized, supporting role in castration resistant cancer progression [XREF_BIBR]."
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