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USP22 activates glucose. 5 / 5
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"The results showed that compared to the control group, USP22 expression knockdown significantly decreased glucose consumption, lactate production, and ATP generation in Sao-2 cells (p < 0.05)."
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"Overexpressing USP22 attenuated deleterious roles of HG in INS-1 cells; but its roles were reversed by up-regulating miR-144-3p."
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"In HG-treated NRK-52E cells, USP22-FLAG overexpression further increased USP22 expression ( Fig. 3 C), downregulated E-cadherin and ZO-1expression, and upregulated Vimentin expression ( Fig. 3 D)."
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"Cell-scratch test results showed that in HG-treated NRK-52E cells, USP22-FLAG overexpression significantly increased cell migration, whereas USP22–C185S-FLAG overexpression had no such function ( Fig.[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Moreover, Usp22 knockdown abolished high glucose stimulation–induced cell cycle arrest, apoptosis, and proinflammatory cytokine production in podocytes [138]."
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