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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

P38 increases the amount of CYLD. 2 / 2

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"The p38 and CYLD pathway is involved in neuronal necroptosis induced by OGD injury and the p38 and CYLD pathway can be activated to increase CYLD protein expression."

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"As p38 directly phosphorylates ELK1, and p38 downstream kinase MAPKAP (MK2) can phosphorylate SRF, we decided to find whether p38 directly or through MK2 induces CYLD gene expression."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...