IndraLab

Statements

USP22 binds MED1. 4 / 4
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"The interaction between the endogenous USP22 and MED1 was further validated in mouse primary iNKT cells because MED1 was detected in anti-USP22 immunoprecipitates but not the normal rabbit IgG controls ( xref ), indicating a possibility that USP22 regulates iNKT cell development through, at least partially, MED1 interaction."
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"Since USP22 interacts with MED1, both of which are transcription coactivators involved in promoting T-bet and IL-2R gene transcription in iNKT cells, we then asked whether MED1 binds to the promoters of T-bet and IL-2Rβ in a USP22-dependent manner, or vice versa."
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"However, assessment of ER stress‐induced changes in MED1 and USP22 binding shows poor correlation (Fig  xref )."
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"USP22 interacts with MED1 in iNKT cells."
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