IndraLab

Statements


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"USP22 was highly expressed in NPC cells and promoted cell viability and proliferation."

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"Moreover, the knockdown of USP22 significantly reduced the viability of CRC cells overexpressing ZRANB1, while the elevation of USP22 in ZRANB1-deficient cells successfully enhanced cell viability to [MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"Under the treatment of Lenvatinib, USP22 knockdown inhibited the cell viability of drug-resistant HCC cells and promoted the apoptosis of drug-resistant cells."

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"Therefore, up-regulation of USP22 expression will lead to abnormal activation of multiple pathways to promote cell survival while down-regulation of USP22 expression can induce cell cycle arrest at G0/G1 phase in different types of cancer cells (Zhang et al., 2008)."

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"Silencing of either USP22 or RNF40 reduced short-term cell viability and clonogenic growth of osteosarcoma cells (Fig. 6A and B)."

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"Similarly , Zhao et al. reported that USP22 depletion suppressed cell survival and proliferation as well as tumor growth and lung metastasis of anaplastic thyroid carcinoma cells25 ."

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"18 In lung cancer, USP22 promotes cell survival and proliferation by upregulating the expression levels of stemness-related markers."