IndraLab

Statements



reach
"Although MG132 prevention of c-Fos and/or c-Jun degradation might account for AP-1 activation, the rapid JNK activation by MG132 is another alternative pathway [15]."

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"In addition, MG132, a proteasome inhibitor, abolished the effect of c-fos depletion by 2-NPPA treatment (XREF_FIG)."