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USP18 activates TNF. 2 / 3
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"As these two signals have been thought to have counter-regulatory roles in psoriasis (39), lower USP18 might promote higher IFN response and thus lower TNF dependence, and vice versa, agreeing with the positive correlation with PASI improvement we observed during the course of etanercept treatment."
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"What’s more, overexpression of USP18 further enhanced the synthesis and secretion of IL-6, TNF-α, IFN-β and CXCL10 after PR8 infection ( Fig. 2 A and B), while inhibition of USP18 had an opposite effe[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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