IndraLab

Statements

OTUB1 inhibits cell population proliferation. 11 / 12
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"The result showed that overexpression of OTUB1 significantly enhanced MCF-7 cell proliferation, whereas OTUB1 depletion decreased the rates of proliferation of MCF-7 cells (XREF_SUPPLEMENTARY)."
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"Knockdown of OTUB1 inhibited cell viability and proliferation, as well as migration and invasion of RCC cells."
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"Additionally, OTUB1 depletion enhances the cytokine production and the proliferation of CD4 T cells (62, 63)."
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"Flow cytometry-based analysis of E18.5 lung tissue revealed that Otub1 deletion increased proliferation of major lung parenchymal and mesenchymal/other non-hematopoietic cell types."
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"OTUB1 knockdown promoted proliferation, inhibited apoptosis and EMT of TGF-β1-treated cells."
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"Consequently, overexpression of Otub1 induces p53 dependent apoptosis and inhibition of cell proliferation whereas knockdown of Otub1 attenuates p53 activation following DNA damage [XREF_BIBR]."
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"Similar to the findings above, OTUB1 knockdown promoted the migration and proliferation of MDA‐MB‐231 (Figure S11A–F) and BT549 (Figure S11G–L) cells."
37608493
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"In fact, cell proliferation of JHH-4 cells was reduced by the depletion of OTUB1 ( Fig. 6 D)."
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"The authors showed OTUB1 knockout increases the proliferation and migration of 4T1 mouse breast cancer cells, which can be counteracted by CCN6 expression."
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"Deletion of OTUB1, concomitant with reduced CCN6 abundance, increased the migration, proliferation and viability of breast cancer cells."
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"In adult mice, deletion of Otub1 also induces proliferation in lung tissue and alveolar hyperventilation."
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