IndraLab

Statements



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"In mast cells, however, Cacna1c knockdown augmented apoptosis after IgE stimulation by disrupting mitochondrial integrity, emphasizing that the maintenance of an appropriate mitochondrial calcium concentration is also essential for cell survival 60."

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"Instead, the deletion of CaV1.2 triggers the apoptotic cell death and loss of approximately half of all adult OPCs from the corpus callosum without affecting the survival of OPCs located in the motor cortex or spinal cord."