IndraLab

Statements

USP53 activates Neoplasm Metastasis. 8 / 8
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"The overexpression of USP53 in TNBC promotes the deubiquitination of CRKL and tumor growth and metastasis in TNBC."
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"USP53 Promotes TNBC Cell Metastasis."
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"Taken together, USP53 promotes metastasis and EMT in TNBC."
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"Here, we show that the deubiquitinase USP53 contributes to tumor growth and metastasis in TNBC."
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"Ubiquitin-specific peptidase 53 (USP53) prevents the inactivation of the NF-κB pathway by reducing ubiquitination of IκBα, thereby further inhibiting ccRCC proliferation and metastasis (Gui et al., 2021)."
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"In this study, we found that USP53 promoted the growth and metastasis of TNBC."
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"USP53 can inhibit the complex of p50 and p65 by deubiquitinizing IκBα, thereby inhibiting the activity of the NF-κB pathway and ultimately inhibiting the proliferation and metastasis of ccRCC [92]."
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"Additionally, the knockdown of USP53 suppressed proliferation, EMT, and metastasis and enhanced the paclitaxel response of TNBC cells."
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