Other Services

This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

USP9X activates PMAIP1. 2 / 2

| 2

➶

reach

"Interestingly, both Usp9X pharmacological inhibition and silencing in MPNST cells elicited a significant increase in ATF3, a typical marker of ER-stress (Fig. 5a,b) and in the pro-apoptotic BH3-only protein Noxa (Fig. 5a,b)."

30478285

➶

reach

"Furthermore, Usp9x expression increased in Noxa knockdown cells, suggesting Noxa may regulate Mcl-1 expression by controlling the expression of Usp9x."

24991768

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...