IndraLab

Statements

OTUD4 activates apoptotic process. 3 / 3
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"What’s more, loss of OTUD4 in Hela cells impaired stress granule formation and promoted apoptosis via activating caspase 3 [20]."
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"Overexpression of OTUD4 increases radiosensitivity of NSCLC cells exhibiting as impaired clonogenic formation ability, enhanced cell cycle arrest and increased cell apoptosis."
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"Expression of HIN-1 in synchronized cells inhibits cell cycle re-entry and the phosphorylation of the retinoblastoma protein (Rb), while in exponentially growing cells HIN-1 induces apoptosis without apparent cell cycle arrest or an effect on Rb phosphorylation."
| PMC