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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

USP42 activates homologous recombination. 4 / 4

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"Altogether, these results indicated that USP42 promoted HR and cellular viability by facilitating DNA-end resection."

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"To understand how USP42 promotes HR, the effects of USP42 depletion with siRNAs on various aspects of HR were investigated."

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"USP42 promotes HR by facilitating DNA-end resection."

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"Depleting USP42 resulted in significantly reduced RAD51 foci formation, suggesting that USP42 functions upstream of RAD51 recruitment to DSB sites and promotes HR."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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IndraLab

Statements

USP42 activates homologous recombination. 4 / 4

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