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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

UCHL3 increases the amount of IFNAR1. 2 / 2

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"These findings suggest that UCHL3 could positively regulate IFNAR1 levels via COPS5 mediated deneddylation of Cullin1, which is consistent with our previous findings that UCHL3 positively regulates IF[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"In summary, our results demonstrate that UCHL3 upregulates the levels of SCF beta-TrCP substrates.We have demonstrated that UCHL3 positively regulated COPS5 levels and IFNAR1 levels."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...