IndraLab

Statements

U0126 inhibits FOS. 5 / 7
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"U0126 inhibits de novo synthesis of c-Fos after T cell stimulation in a dose dependent manner."
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"EGF increased p-ERK1/2, c-Fos, c-Jun, and p-c-Jun levels, which were inhibited by U0126."
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"Intrathecal pretreatment with U0126 (an inhibitor of MEK) reversed the pain behaviors (Fig. 1D and E), the increase of spinal p-ERK1/2 (Fig. 1F) and the increase in spinal c-Fos protein, a marker for neuronal activation (Fig. 1G and H)."
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"The use of U0126, an ERK inhibitor, effectively suppressed HGF mediated induction of Egr1, c-Fos, GDNF, and Lif as well as SC migration."
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"Zta protein production was stimulated by TPA at 6h and maintained at a high level until 24h, ERK phosphorylation was induced by TPA from 1h to 12h, c-Jun phosphorylation was induced by TPA at 1h and increased in a time dependent manner, whereas c-Fos phosphorylation was up-regulated by TPA from 3h to 12h, however, TPA induced production of Zta and phosphorylation of ERK, c-Jun, and c-Fos were repressed by U0126 (XREF_FIG)."
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